ANSWER
Identifieur interne : 001A34 ( Main/Exploration ); précédent : 001A33; suivant : 001A35ANSWER
Auteurs :Source :
- Thorax [ 0040-6376 ] ; 2008-12.
English descriptors
- Teeft :
- Aberrant protein, Active production, Airspace, Airspace consolidation, Airspaces show, Airway, Alveolar macrophages, Alveolar macrophages pattern, Alveolar type, Alveolar walls, Alveolitis, Amin, Aminoacidic, Aminoacidic substitution, Antigen presentation, Antimalarial, Antimalarial agents, Balt, Balt hyperplasia, Baughman, Biol, Biol chem rosen, Biosynthesis, Cell infiltration, Cellular injury, Cellular processing, Centile, Chem, Cholesterol clefts, Chronic hypoxaemia, Clinical condition, Clinical improvement, Clubbing, Congenital surfactant deficiency, Continuous hydroxychloroquine treatment, Cuboidal, Cuboidal metaplastic changes, Cyanosis, December, Deficiency mutations, Diffuse hypercellularity, Diffuse phagocytosis, Digital clubbing, Disventilation, Endogenous lipidic alveolitis, Engl, Exact mechanism, Focal overdistension, Gene cause, Genetic analysis, Genetic background, Griese, Hartl, Heterozygous mutations, Histiocyte, Hydrophobic protein, Hydroxychloroquine, Hydroxychloroquine treatment, Hypoxaemia, Important role, Inflammatory mediators, Interfamilial, Interfamilial phenotypic variability, Interleukin, Interstitial lung disease, Intracellular, Intracellular processing, Lamellar, Lamellar bodies, Lipidic, Lung biopsy, Lung damage, Lung disease, Lung infections, Lymphocyte, Macrophage, Macrophagic, Macrophagic alveolitis, Mediator, Metaplastic, Mutation, Open lung biopsy, Overdistension, Oxygen saturation, Pathway, Patients hydroxychloroquine treatment, Pediatr, Pediatr figure, Phagocytosis, Phenotype, Phenotypic, Physical activity, Possible benefit, Profound effects, Protein degradation pathway, Pulmonary inflammation, Pulmonary puzzle answer, Receptor, Recurrent lung infection, Regular biosynthesis, Regular pathway, Respir, Respiratory rate, Septal, Severe alveolar septal, Significant fibrosis, Stabilisation, Surfactant, Surfactant protein, Surfactant protein deficiency, Thorax, Thorax december, Thorax references, Tolllike, Tolllike receptors, Undiagnosed, Upper airway infection.
Url:
DOI: 10.1136/thx.2007.092650a
Affiliations:
Links toward previous steps (curation, corpus...)
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- to stream Istex, to step Curation: 000A08
- to stream Istex, to step Checkpoint: 000940
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- to stream Main, to step Curation: 001A34
Le document en format XML
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<profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Aberrant protein</term>
<term>Active production</term>
<term>Airspace</term>
<term>Airspace consolidation</term>
<term>Airspaces show</term>
<term>Airway</term>
<term>Alveolar macrophages</term>
<term>Alveolar macrophages pattern</term>
<term>Alveolar type</term>
<term>Alveolar walls</term>
<term>Alveolitis</term>
<term>Amin</term>
<term>Aminoacidic</term>
<term>Aminoacidic substitution</term>
<term>Antigen presentation</term>
<term>Antimalarial</term>
<term>Antimalarial agents</term>
<term>Balt</term>
<term>Balt hyperplasia</term>
<term>Baughman</term>
<term>Biol</term>
<term>Biol chem rosen</term>
<term>Biosynthesis</term>
<term>Cell infiltration</term>
<term>Cellular injury</term>
<term>Cellular processing</term>
<term>Centile</term>
<term>Chem</term>
<term>Cholesterol clefts</term>
<term>Chronic hypoxaemia</term>
<term>Clinical condition</term>
<term>Clinical improvement</term>
<term>Clubbing</term>
<term>Congenital surfactant deficiency</term>
<term>Continuous hydroxychloroquine treatment</term>
<term>Cuboidal</term>
<term>Cuboidal metaplastic changes</term>
<term>Cyanosis</term>
<term>December</term>
<term>Deficiency mutations</term>
<term>Diffuse hypercellularity</term>
<term>Diffuse phagocytosis</term>
<term>Digital clubbing</term>
<term>Disventilation</term>
<term>Endogenous lipidic alveolitis</term>
<term>Engl</term>
<term>Exact mechanism</term>
<term>Focal overdistension</term>
<term>Gene cause</term>
<term>Genetic analysis</term>
<term>Genetic background</term>
<term>Griese</term>
<term>Hartl</term>
<term>Heterozygous mutations</term>
<term>Histiocyte</term>
<term>Hydrophobic protein</term>
<term>Hydroxychloroquine</term>
<term>Hydroxychloroquine treatment</term>
<term>Hypoxaemia</term>
<term>Important role</term>
<term>Inflammatory mediators</term>
<term>Interfamilial</term>
<term>Interfamilial phenotypic variability</term>
<term>Interleukin</term>
<term>Interstitial lung disease</term>
<term>Intracellular</term>
<term>Intracellular processing</term>
<term>Lamellar</term>
<term>Lamellar bodies</term>
<term>Lipidic</term>
<term>Lung biopsy</term>
<term>Lung damage</term>
<term>Lung disease</term>
<term>Lung infections</term>
<term>Lymphocyte</term>
<term>Macrophage</term>
<term>Macrophagic</term>
<term>Macrophagic alveolitis</term>
<term>Mediator</term>
<term>Metaplastic</term>
<term>Mutation</term>
<term>Open lung biopsy</term>
<term>Overdistension</term>
<term>Oxygen saturation</term>
<term>Pathway</term>
<term>Patients hydroxychloroquine treatment</term>
<term>Pediatr</term>
<term>Pediatr figure</term>
<term>Phagocytosis</term>
<term>Phenotype</term>
<term>Phenotypic</term>
<term>Physical activity</term>
<term>Possible benefit</term>
<term>Profound effects</term>
<term>Protein degradation pathway</term>
<term>Pulmonary inflammation</term>
<term>Pulmonary puzzle answer</term>
<term>Receptor</term>
<term>Recurrent lung infection</term>
<term>Regular biosynthesis</term>
<term>Regular pathway</term>
<term>Respir</term>
<term>Respiratory rate</term>
<term>Septal</term>
<term>Severe alveolar septal</term>
<term>Significant fibrosis</term>
<term>Stabilisation</term>
<term>Surfactant</term>
<term>Surfactant protein</term>
<term>Surfactant protein deficiency</term>
<term>Thorax</term>
<term>Thorax december</term>
<term>Thorax references</term>
<term>Tolllike</term>
<term>Tolllike receptors</term>
<term>Undiagnosed</term>
<term>Upper airway infection</term>
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